Document Type

Journal Article

Publication Title

Epigenetics

Volume

18

Issue

1

First Page

2211361

PubMed ID

37233989

Publisher

Taylor & Francis

School

School of Medical and Health Sciences

RAS ID

60153

Funders

National Health and Medical Research Council / Edith Cowan University

Further funding information https://doi.org/10.1080/15592294.2023.2211361

Grant Number

NHMRC Numbers : 572613, 403981, 1059711, 1053384, 2001203, 353514, 403981, 572613, 1059711,

Grant Link

http://purl.org/au-research/grants/nhmrc/572613

http://purl.org/au-research/grants/nhmrc/1059711

http://purl.org/au-research/grants/nhmrc/1053384

http://purl.org/au-research/grants/nhmrc/2001203

http://purl.org/au-research/grants/nhmrc/572613

http://purl.org/au-research/grants/nhmrc/1059711

Comments

Keshawarz, A., Joehanes, R., Ma, J., Lee, G. Y., Costeira, R., Tsai, P. C., . . . Levy, D. (2023). Dietary and supplemental intake of vitamins C and E is associated with altered DNA methylation in an epigenome-wide association study meta-analysis. Epigenetics, 18(1), article 2211361.

https://doi.org/10.1080/15592294.2023.2211361

Abstract

BACKGROUND: Dietary intake of antioxidants such as vitamins C and E protect against oxidative stress, and may also be associated with altered DNA methylation patterns. METHODS: We meta-analysed epigenome-wide association study (EWAS) results from 11,866 participants across eight population-based cohorts to evaluate the association between self-reported dietary and supplemental intake of vitamins C and E with DNA methylation. EWAS were adjusted for age, sex, BMI, caloric intake, blood cell type proportion, smoking status, alcohol consumption, and technical covariates. Significant results of the meta-analysis were subsequently evaluated in gene set enrichment analysis (GSEA) and expression quantitative trait methylation (eQTM) analysis. RESULTS: In meta-analysis, methylation at 4,656 CpG sites was significantly associated with vitamin C intake at FDR ≤ 0.05. The most significant CpG sites associated with vitamin C (at FDR ≤ 0.01) were enriched for pathways associated with systems development and cell signalling in GSEA, and were associated with downstream expression of genes enriched in the immune response in eQTM analysis. Furthermore, methylation at 160 CpG sites was significantly associated with vitamin E intake at FDR ≤ 0.05, but GSEA and eQTM analysis of the top most significant CpG sites associated with vitamin E did not identify significant enrichment of any biological pathways investigated. CONCLUSIONS: We identified significant associations of many CpG sites with vitamin C and E intake, and our results suggest that vitamin C intake may be associated with systems development and the immune response.

DOI

10.1080/15592294.2023.2211361

Creative Commons License

Creative Commons License
This work is licensed under a Creative Commons Public Domain Mark 1.0.

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