Document Type

Journal Article

Publication Title

GeroScience

Volume

46

First Page

5911

Last Page

5923

Publisher

Springer

School

School of Medical and Health Sciences

RAS ID

69883

Funders

Open Access funding enabled and organized by CAUL and its Member Institutions / National Health and Medical Research Council

Grant Number

NHMRC Number : GNT1097105

Grant Link

http://purl.org/au-research/grants/nhmrc/1097105

Comments

Sewell, K. R., Rainey-Smith, S. R., Pedrini, S., Peiffer, J. J., Sohrabi, H. R., Taddei, K., . . . Brown, B. M. (2024). The impact of exercise on blood-based biomarkers of Alzheimer’s disease in cognitively unimpaired older adults. GeroScience, 46, 5911-5923. https://doi.org/10.1007/s11357-024-01130-2

Abstract

Physical activity is a promising preventative strategy for Alzheimer’s disease: it is associated with lower dementia risk, better cognition, greater brain volume and lower brain beta-amyloid. Blood-based biomarkers have emerged as a low-cost, non-invasive strategy for detecting preclinical Alzheimer’s disease, however, there is limited literature examining the effect of exercise (a structured form of physical activity) on blood-based biomarkers. The current study investigated the influence of a 6-month exercise intervention on levels of plasma beta-amyloid (A 42, A 40, A 42/40), phosphorylated tau (p-tau181), glial fibrillary acidic protein (GFAP) and neurofilament light (NfL) chain in cognitively unimpaired older adults, and as a secondary aim, whether blood-based biomarkers related to cognition. Ninety-nine community-dwelling older adults (69.1 ± 5.2) were allocated to an inactive control, or to moderate or high intensity exercise groups where they cycled twice weekly for six months. At baseline and six months (post-intervention), fasted blood was collected and analysed using single molecule array (SIMOA) assays, and cognition was assessed. Results demonstrated no change in levels of any plasma biomarker from pre- to post-intervention. At baseline, higher NfL was associated with poorer cognition ( = -0.33, SE = 0.13, adjusted p =.042). Exploratory analyses indicated higher cardiorespiratory fitness was associated with higher NfL and GFAP levels in apolipoprotein E (APOE) 4 non-carriers compared to 4 carriers (NfL, = -0.43, SE = 0.19, p =.029; GFAP, = -0.41, SE = 0.20, p =.044), though this association was mediated by body mass index (BMI). These results highlight the importance of considering BMI in analysis of blood-based biomarkers, especially when investigating differences between APOE ε4 carriers and non-carriers. Our results also indicate that longer follow-up periods may be required to observe exercise-induced change in blood-based biomarkers.

DOI

10.1007/s11357-024-01130-2

Creative Commons License

Creative Commons Attribution 4.0 License
This work is licensed under a Creative Commons Attribution 4.0 License.

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