Author Identifier

Chunbin Zhou

http://orcid.org/0000-0001-9367-0756

Date of Award

2024

Document Type

Thesis - ECU Access Only

Publisher

Edith Cowan University

Degree Name

Doctor of Philosophy

School

School of Medical and Health Sciences

First Supervisor

Wei Wang

Second Supervisor

Hu Wang

Third Supervisor

Lois Balmer

Fourth Supervisor

Manshu Song

Abstract

Osteosarcoma is the most prevalent primary bone cancer, posing challenges due to its high aggressiveness and propensity for chemotherapy resistance. Circular RNAs (circRNAs) can function as competing endogenous RNAs (ceRNAs), playing a crucial role in cancer progression, including invasion, metastasis and chemotherapy resistance. The expression profile and role of circRNAs in osteosarcoma remain unclear. Thus, my PhD identified dysregulated circRNAs and investigated their potential regulatory mechanisms in the pathogenesis of osteosarcoma.

An updated systematic review and meta-analysis was conducted based on articles published from January 2010 to September 2023 across eight databases (PubMed, EMBASE, Cochrane Library, Web of Science, Wanfang, Sinomed, CNKI, Vip) to identify consistently reported circRNAs as potential biomarkers in osteosarcoma. A total of 58 dysregulated circRNAs (52 upregulated and 6 downregulated) were identified, with 11 of them exhibiting dysregulation in both osteosarcoma tissues and cell lines. Among the dysregulated circRNAs, hsa_circ_0005721 showed potential as a circulating biomarker in osteosarcoma, and hsa_circ_0001821 was the most reported circRNA.

The in vitro experiments were performed to explore the potential mechanism underlying hsa_circ_0001821, also known as circPVT1. CircPVT1 was highly expressed in osteosarcoma cells and facilitated cells’ capability of migration and invasion. Mechanistically, circPVT1 modulated the expression of HAVCR2 by acting as a miR-490-5p sequester, in which overexpression of miR-490-5p resulted in a significant reduction of migration and invasion of osteosarcoma cells, whereas HAVCR2 overexpression had the opposite effect.

High-throughput RNA sequencing was employed on doxorubicin sensitivity and resistant osteosarcoma cell lines. The bioinformatics analysis revealed that 163 circRNAs exhibited differential expression, with 57 being upregulated and 106 downregulated in doxorubicin resistant osteosarcoma cells. Based on the prediction from various databases, a ceRNA network consisting of 6 circRNAs, 8 miRNAs and 16 mRNAs was constructed, including 9 circRNA-miRNA-mRNA axes all sharing miR-188-3p, which were deemed potentially crucial in the development of doxorubicin resistance in osteosarcoma.

My PhD project identified pivotal roles for circRNAs in the pathogenesis of osteosarcoma and expanded comprehension of circRNA expression profiles and their underlying regulatory mechanisms in osteosarcoma.

DOI

10.25958/29gt-vt41

Access Note

Access to this thesis is embargoed until 26th November 2027

Available for download on Friday, November 26, 2027

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