Author Identifier

Kevin Mendez

http://orcid.org/0000-0002-8832-2607

Date of Award

2024

Document Type

Thesis

Publisher

Edith Cowan University

Degree Name

Doctor of Philosophy

School

School of Science

First Supervisor

Stacey Reinke

Second Supervisor

David Broadhurst

Third Supervisor

Jessica Lasky-Su

Fourth Supervisor

Rachel Kelly

Abstract

Asthma is a heterogeneous lung disease characterized by diverse clinical presentations, pathophysiological mechanisms, and treatment responses. Understanding the varied clinical presentations, known as asthma phenotypes, aids in elucidating the underlying biological processes and improving treatment strategies. These phenotypes encompass measures of airway hyperresponsiveness, atopy, lung function, and inflammation, driven by the interplay of genetic and environmental factors. Investigating and characterizing the metabolomic profiles —reflecting both genetic and environmental influences— associated with distinct asthma phenotypes may provide new insights into disease mechanisms and potential therapeutic targets.

This thesis is structured around three specific aims: (1) identify metabolomic associations within and across standard asthma phenotypes; (2) explore metabolomic associations with lung function trajectory (LTF) patterns in asthmatics; and (3) develop and investigate electronic medical record (EMR)-based metrics of lung health decline using metabolomics in asthmatics.

For Aim 1, this thesis utilizes two pediatric asthmatic cohorts with detailed clinical presentations and untargeted metabolomic data: the Childhood Asthma Management Program (CAMP, n=953) and the Genetic Epidemiology of Asthma in Costa Rica Study (GACRS, n=1155). For Aim 2, the thesis focuses on a subset of the CAMP cohort (n=660) where lung function trajectory (LFT) patterns had been characterized, grouping patients into: normal growth (NG), early decline (ED), reduced growth (RG), and reduced growth with early decline (RG/ED). For Aim 3, this thesis incorporates previously extracted data from two EMR-linked large-scale cohorts from Mass General Brigham (MGB): the Omic Determinants of Longitudinal Lung Function cohort (ODOLLFA, n=511), consisting of asthmatic individuals, and the MGB-Aging Biobank Cohort (MGB-ABC, n=488) from the general population. By utilizing longitudinal lung function data extracted from the EMR, more precise metrics of lung health can be developed for asthmatics. Integrating these metrics with other data, including previously developed biological aging biomarkers (e.g., epigenetic aging clocks) and untargeted metabolomic data, enables a comprehensive analysis.

This thesis leverages data from these four large-scale cohorts to achieve its aims. The findings underscore the importance of asthma phenotype research and highlight the potential of metabolomics to provide clinically relevant insights into the understanding and management of asthma.

DOI

10.25958/vm3b-vf08

Access Note

Access to this thesis is embargoed until 20th December 2026

Available for download on Sunday, December 20, 2026

Included in

Chemistry Commons

Share

 
COinS